Bacterial Pathogenesis, General Microbiology
Our laboratory is focused on questions related to pathogen evolution, gene expression, and adaptation to different environments. We are especially interested in understanding how bacterial pathogens disrupt, avoid, or otherwise compromise the innate immune systems of their hosts. We primarily work with bacteria in the genus Yersinia, which includes Y. pestis, the cause of bubonic plague, as well as Y. pseudotuberculosis and Y. enterocolitica, which cause food-borne illnesses.
NIH/NIAID, Rocky Mountain Laboratories, Bacterial Pathogenesis, Visiting fellow (2006)
University of Calgary, Bacterial Pathogenesis, Ph.D. (2003)
University of Lethbridge, Lethbridge AB Canada, Biochemistry, B.Sc. (1999)
Siebach TW, Griffitts JS, Wilson E, Erickson DL. 2018. Genome-Wide Identification of Fitness Factors in Mastitis-Associated Escherichia coli. 84(2). doi:10.1128/AEM.02190-17
Russell CW, Johnson KL, Mortensen RD, Erickson DL. 2012. Gene expression analysis of Xenopsylla cheeps (Siphonaptera: Pulicidae) suggests a role for reactive oxygen species in response to Yersinia pestis infection. Journal of Medical Entomology. 49(2):364-70.
Russell CW, Johnson KL, Hileman T, Stewart RM. 2011. PhoP and OxyR transcriptional regulators contribute to Yersinia pestis virulence and survival within Galleria mellonella. Microbial Pathogenesis. 51(6):389-95.
Green C, Day B, Stevens KM, Erickson DL, Woods DE, Storey DG. 2011. The stringent response is essential for Pseudomonas aeruginosa virulence in a chronic infection of the rat lung and the Drosophila melanogaster feeding model of infection. Infection and Immunity. 79(10):4094-104.