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David Erickson

Associate Professor
Microbiology & Molecular Biology

3133 LSB
Provo, UT 84602

Teaching Interests

  • Bacterial Pathogenesis, General Microbiology

Research Interests

Our laboratory is focused on questions related to pathogen evolution, gene expression, and adaptation to different environments. We are especially interested in understanding how bacterial pathogens disrupt, avoid, or otherwise compromise the innate immune systems of their hosts. We primarily work with bacteria in the genus Yersinia, which includes Y. pestis, the cause of bubonic plague, as well as Y. pseudotuberculosis and Y. enterocolitica, which cause food-borne illnesses.

Courses Taught

Winter 2019

  • MMBIO 294R: Mentored Research Section 006
  • MMBIO 364: Bacterial Pathogenesis Section 001
  • MMBIO 365: Bacterial Pathogenesis Lab Section 001
  • MMBIO 695R: Research Section 006

Fall 2018

  • MMBIO 390R: Readings in Molecular Biology Section 001
  • MMBIO 490R: Molecular Biology Seminar Section 001
  • MMBIO 494R: Advanced Mentored Research Section 006
  • MMBIO 516: Bacteria-Host Interactions Section 001
  • MMBIO 691R: Graduate Seminar Section 001
  • MMBIO 695R: Research Section 006

Summer 2018

  • MMBIO 695R: Research Section 006

Spring 2018

  • MMBIO 494R: Mentored Research Section 006


Journal Articles

Olson MA, Siebach TW, Griffitts JS, Wilson E, Erickson DL. 2018. Genome-Wide Identification of Fitness Factors in Mastitis-Associated Escherichia coli. 84(2). doi:10.1128/AEM.02190-17

Zhou W, Russell CW, Johnson KL, Mortensen RD, Erickson DL. 2012. Gene expression analysis of Xenopsylla cheeps (Siphonaptera: Pulicidae) suggests a role for reactive oxygen species in response to Yersinia pestis infection. Journal of Medical Entomology. 49(2):364-70.

Erickson DL, Russell CW, Johnson KL, Hileman T, Stewart RM. 2011. PhoP and OxyR transcriptional regulators contribute to Yersinia pestis virulence and survival within Galleria mellonella. Microbial Pathogenesis. 51(6):389-95.

Vogt SL, Green C, Day B, Stevens KM, Erickson DL, Woods DE, Storey DG. 2011. The stringent response is essential for Pseudomonas aeruginosa virulence in a chronic infection of the rat lung and the Drosophila melanogaster feeding model of infection. Infection and Immunity. 79(10):4094-104.